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Body fat and dairy product intake in lactase persistent and non-persistent children and adolescents
Örebro universitet, Hälsoakademin.
Unit Prevent Nutr, Dept Biosci & Nutr, Karolinska Inst, Huddinge, Sweden; Sch Biol Sci, Dublin Inst Technol, Dublin, Ireland.
Örebro University Hospital, Örebro, Sweden.
Örebro universitet, Hälsoakademin.ORCID-id: 0000-0001-8828-9299
Vise andre og tillknytning
2010 (engelsk)Inngår i: Food & Nutrition Research, ISSN 1654-6628, E-ISSN 1654-661X, Vol. 54, artikkel-id 5141Artikkel i tidsskrift (Fagfellevurdert) Published
Abstract [en]

Background: Lactase non-persistent (LNP) individuals may be lactose intolerant and therefore on a more restricted diet concerning milk and milk products compared to lactase persistent (LP) individuals. This may have an impact on body fat mass.

Objective This study examines if LP and LNP children and adolescents, defined by genotyping for the LCT-13910 C > T polymorphism, differ from each other with regard to milk and milk product intake, and measures of body fat mass.

Design: Children (n=298, mean age 9.6 years) and adolescents (n=386, mean age 15.6 years), belonging to the Swedish part of the European Youth Heart Study, were genotyped for the LCT-13910 C > T polymorphism. Dietary intakes of reduced and full-fat dairy varieties were determined.

Results: LNP (CC genotype) subjects consumed less milk, soured milk and yoghurt compared to LP (CT/TT genotype) subjects (p<0.001). Subsequent partitioning for age group attenuated this observation (p=0.002 for children and p=0.023 in adolescents). Six subjects were reported by parents to be 'lactose intolerant', none of whom were LNP. LNP children and adolescents consumed significantly less reduced fat milk and milk products than LP children and adolescents (p=0.009 for children and p=0.001 for adolescents).

Conclusions: We conclude that LP is linked to an overall higher milk and dairy intake, but is not linked to higher body fat mass in children and adolescents.

sted, utgiver, år, opplag, sider
Järfälla, Sweden: Co-action Publishing , 2010. Vol. 54, artikkel-id 5141
HSV kategori
Forskningsprogram
Medicin
Identifikatorer
URN: urn:nbn:se:oru:diva-15350DOI: 10.3402/fnr.v54i0.5141ISI: 000208683500006PubMedID: 20585563Scopus ID: 2-s2.0-77955476574OAI: oai:DiVA.org:oru-15350DiVA, id: diva2:412191
Tilgjengelig fra: 2011-04-21 Laget: 2011-04-21 Sist oppdatert: 2018-04-23bibliografisk kontrollert
Inngår i avhandling
1. Lactase Persistence and Lactase Non-Persistence: Prevalence, influence on body fat, body height, and relation to the metabolic syndrome
Åpne denne publikasjonen i ny fane eller vindu >>Lactase Persistence and Lactase Non-Persistence: Prevalence, influence on body fat, body height, and relation to the metabolic syndrome
2010 (engelsk)Doktoravhandling, med artikler (Annet vitenskapelig)
sted, utgiver, år, opplag, sider
Örebro: Örebro universitet, 2010. s. 45
Serie
Örebro Studies in Medicine, ISSN 1652-4063 ; 48
HSV kategori
Forskningsprogram
Medicin
Identifikatorer
urn:nbn:se:oru:diva-11854 (URN)978-91-7668-767-3 (ISBN)
Disputas
2010-12-15, Wilandersalen, Universitetssjukhuset Örebro, Fakultetsgatan 1, 701 82 Örebro, 09:00
Opponent
Veileder
Tilgjengelig fra: 2010-09-17 Laget: 2010-09-17 Sist oppdatert: 2017-10-17bibliografisk kontrollert

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